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Tuscan tourist center

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Want to know more about Cary? For the 1st time, a visitor center is ready to welcome you

Walking into the Cary Visitor Center, you’ll find the 1976 municipal flag that flew over the town until 2001.

You’ll see books by local writers and bricks from the old Page-Walker Hotel, built in 1868, which serves as the town’s arts and history center.

If your family has lived here long enough, you may even spot your parents in a Cary High School yearbook.

As the town has grown to over 180,000 people, there has been a push for new developments, shopping centers and restaurants. Yet, there has never been a physical location to go to when seeking general information or to request a tour.

The Cary Visitor Center has opened at 107 W. Chatham St. this month, thanks to two Cary natives who wanted to change that.

“I think it’s really an interesting unifier where people, regardless of their legacy or their time in Cary, are coming back to one place to see it in a new way,” said Katherine Loflin, a town historian and business owner.

Though you can find recommendations, the Cary Visitor Center is more than maps and dinner suggestions.

It presents Cary “in a way that makes people want to fall in love with it,” Loflin said.

‘The new merging with the old’

In February 91-year-old Ralph Ashworth and his son’s decision to close Ashworth Drugs, a neighborhood pharmacy and lunch spot on West Chatham Street, sparked the idea for the visitor center.

Their decision left many residents reflecting on changes in downtown Cary, such as new restaurants and nightlife, a future apartment building , and, recently, the $68 million Downtown Cary Park. Many felt the places they loved would soon disappear.

“I thought to myself, this is a moment we need to step up for Cary because (the closing) was disorienting,” Loflin said. “And I knew a government-operated visitor center in Cary was going to still be elusive for a while.”

Loflin, who leads paranormal tours and stages plays around town under her business, The City Doctor Productions, and Leslie Douglas, the owner of Douglas Realty Group in downtown Cary, opened the visitor center in the realty group’s building.

“I remember when the only reason you went downtown Cary was to get a (sandwich) from Mr. Ashworth,” Douglas said. “I think you can embrace the new and the change, at the same time celebrating the history. I wanted this to be a place where the new merges with the old and we appreciate where we come from.”

What’s in the Cary Visitor Center?

Cary residents have donated books, maps, flags and commemorative antiques to the center.

Ashworth, who had been in business 67 years, donated awards the business had won, the awning that hung over the lunch counter, and original coins from Cary’s centennial celebration.

Other donated items are original art by Jerry Miller, the Cary artist behind the town’s Lazy Daze Arts & Crafts Festival that began in 1977; railroad spikes; and facts about Henry Adams, who played a role in integrating Cary High School.

There are also replicas of drums from the Tuscarora Native American tribe made by Eagle Scouts in the 1950s.

“If you thought you knew everything about Cary, you might want to come in and check us out,” Douglas said.

The Cary Visitor Center is open Monday to Friday from 9 a.m. to 5 p.m. and on weekends from 10 a.m. to 4 p.m.

©2024 Raleigh News & Observer. Visit newsobserver.com. Distributed by Tribune Content Agency, LLC.

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Visitor Centers

Maybe you planned your trip to the minute, or maybe you’re just winging it. Either way, be sure to fit in a stop to your nearest Arizona Visitor Information Center.

Why stop at an Arizona Visitor Information Center?

For starters, you can:

• Pick up our latest Official State Travel Guide, maps and other brochures from local businesses and travel partners to spark more ideas for your trip.
• Get the inside scoop from locals on what’s happening in the area, restaurants to try, places to stay and attractions to put on your must-see list.
• Learn which nearby sites or parks need permits (and how to get one) and which are free to visit.
• Find discount coupons or “passport books” with special offers and deals for visitors and tourists.

Important details to know: Most Arizona Information Visitor Centers are open Monday – Friday, 9 a.m. - 5 p.m. but check each listing as hours may vary. Centers are closed on all major and federal holidays, including Easter Sunday, Independence Day, Labor Day, Thanksgiving and Christmas.

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Apache Junction Area Chamber of Commerce

In the shadow of the majestic Superstition Mountains, Apache Junction is a desert playground with a historic past. The Chamber of Commerce...

567 W Apache Trail Apache Junction, Arizona 85120

Arizona Heritage Center at Papago Park

Tucked against the beautiful Papago Park preserve, the Arizona Heritage Center is an oasis in the desert. In the heart of the Valley of the...

1300 N College Ave Tempe, Arizona 85281

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Celebrate the proud history of southern Arizona by visiting the historic Josias- Joesler-designed Arizona History Museum in Tucson. Cruise...

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In the heart of Cochise County, Benson proudly calls itself home to the living caves of Kartchner Caverns State Park and the peaceful Holy...

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Find out what's happening in this quirky, artsy, historic town in southern Arizona with a stop at the visitor center, located within the...

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From swimming and boating to hiking and camping, you can do it all at Buckskin Mountain State Park. This park has some of the finest views...

5476 No. US Hwy 95 Parker, Arizona 85344

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Camp Verde's Visitor Center shares space with the Camp Verde Historical Society Museum--down the street from the Verde Valley Archaeology...

435 S Main St Camp Verde, Arizona 86322

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575 N Marshall St Casa Grande, Arizona 85122

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Two key brain systems are central to psychosis, Stanford Medicine-led study finds

When the brain has trouble filtering incoming information and predicting what’s likely to happen, psychosis can result, Stanford Medicine-led research shows.

April 11, 2024 - By Erin Digitale

People with psychosis have trouble filtering relevant information (mesh funnel) and predicting rewarding events (broken crystal ball), creating a complex inner world. Emily Moskal

Inside the brains of people with psychosis, two key systems are malfunctioning: a “filter” that directs attention toward important external events and internal thoughts, and a “predictor” composed of pathways that anticipate rewards.

Dysfunction of these systems makes it difficult to know what’s real, manifesting as hallucinations and delusions. 

The findings come from a Stanford Medicine-led study , published April 11 in  Molecular Psychiatry , that used brain scan data from children, teens and young adults with psychosis. The results confirm an existing theory of how breaks with reality occur.

“This work provides a good model for understanding the development and progression of schizophrenia, which is a challenging problem,” said lead author  Kaustubh Supekar , PhD, clinical associate professor of psychiatry and behavioral sciences.

The findings, observed in individuals with a rare genetic disease called 22q11.2 deletion syndrome who experience psychosis as well as in those with psychosis of unknown origin, advance scientists’ understanding of the underlying brain mechanisms and theoretical frameworks related to psychosis.

During psychosis, patients experience hallucinations, such as hearing voices, and hold delusional beliefs, such as thinking that people who are not real exist. Psychosis can occur on its own and isa hallmark of certain serious mental illnesses, including bipolar disorder and schizophrenia. Schizophrenia is also characterized by social withdrawal, disorganized thinking and speech, and a reduction in energy and motivation.

It is challenging to study how schizophrenia begins in the brain. The condition usually emerges in teens or young adults, most of whom soon begin taking antipsychotic medications to ease their symptoms. When researchers analyze brain scans from people with established schizophrenia, they cannot distinguish the effects of the disease from the effects of the medications. They also do not know how schizophrenia changes the brain as the disease progresses. 

To get an early view of the disease process, the Stanford Medicine team studied young people aged 6 to 39 with 22q11.2 deletion syndrome, a genetic condition with a 30% risk for psychosis, schizophrenia or both. 

Kaustubh Supekar

Brain function in 22q11.2 patients who have psychosis is similar to that in people with psychosis of unknown origin, they found. And these brain patterns matched what the researchers had previously theorized was generating psychosis symptoms.

“The brain patterns we identified support our theoretical models of how cognitive control systems malfunction in psychosis,” said senior study author  Vinod Menon , PhD, the Rachael L. and Walter F. Nichols, MD, Professor; a professor of psychiatry and behavioral sciences; and director of the  Stanford Cognitive and Systems Neuroscience Laboratory .

Thoughts that are not linked to reality can capture the brain’s cognitive control networks, he said. “This process derails the normal functioning of cognitive control, allowing intrusive thoughts to dominate, culminating in symptoms we recognize as psychosis.”

Cerebral sorting  

Normally, the brain’s cognitive filtering system — aka the salience network — works behind the scenes to selectively direct our attention to important internal thoughts and external events. With its help, we can dismiss irrational thoughts and unimportant events and focus on what’s real and meaningful to us, such as paying attention to traffic so we avoid a collision.

The ventral striatum, a small brain region, and associated brain pathways driven by dopamine, play an important role in predicting what will be rewarding or important. 

For the study, the researchers assembled as much functional MRI brain-scan data as possible from young people with 22q11.2 deletion syndrome, totaling 101 individuals scanned at three different universities. (The study also included brain scans from several comparison groups without 22q11.2 deletion syndrome: 120 people with early idiopathic psychosis, 101 people with autism, 123 with attention deficit/hyperactivity disorder and 411 healthy controls.) 

The genetic condition, characterized by deletion of part of the 22nd chromosome, affects 1 in every 2,000 to 4,000 people. In addition to the 30% risk of schizophrenia or psychosis, people with the syndrome can also have autism or attention deficit hyperactivity disorder, which is why these conditions were included in the comparison groups.

The researchers used a type of machine learning algorithm called a spatiotemporal deep neural network to characterize patterns of brain function in all patients with 22q11.2 deletion syndrome compared with healthy subjects. With a cohort of patients whose brains were scanned at the University of California, Los Angeles, they developed an algorithmic model that distinguished brain scans from people with 22q11.2 deletion syndrome versus those without it. The model predicted the syndrome with greater than 94% accuracy. They validated the model in additional groups of people with or without the genetic syndrome who had received brain scans at UC Davis and Pontificia Universidad Católica de Chile, showing that in these independent groups, the model sorted brain scans with 84% to 90% accuracy.

The researchers then used the model to investigate which brain features play the biggest role in psychosis. Prior studies of psychosis had not given consistent results, likely because their sample sizes were too small. 

Vinod Menon

Comparing brain scans from 22q11.2 deletion syndrome patients who had and did not have psychosis, the researchers showed that the brain areas contributing most to psychosis are the anterior insula (a key part of the salience network or “filter”) and the ventral striatum (the “reward predictor”); this was true for different cohorts of patients.

In comparing the brain features of people with 22q11.2 deletion syndrome and psychosis against people with psychosis of unknown origin, the model found significant overlap, indicating that these brain features are characteristic of psychosis in general.

A second mathematical model, trained to distinguish all subjects with 22q11.2 deletion syndrome and psychosis from those who have the genetic syndrome but without psychosis, selected brain scans from people with idiopathic psychosis with 77.5% accuracy, again supporting the idea that the brain’s filtering and predicting centers are key to psychosis.

Furthermore, this model was specific to psychosis: It could not classify people with idiopathic autism or ADHD.

“It was quite exciting to trace our steps back to our initial question — ‘What are the dysfunctional brain systems in schizophrenia?’ — and to discover similar patterns in this context,” Menon said. “At the neural level, the characteristics differentiating individuals with psychosis in 22q11.2 deletion syndrome are mirroring the pathways we’ve pinpointed in schizophrenia. This parallel reinforces our understanding of psychosis as a condition with identifiable and consistent brain signatures.” However, these brain signatures were not seen in people with the genetic syndrome but no psychosis, holding clues to future directions for research, he added.

Applications for treatment or prevention

In addition to supporting the scientists’ theory about how psychosis occurs, the findings have implications for understanding the condition — and possibly preventing it.

“One of my goals is to prevent or delay development of schizophrenia,” Supekar said. The fact that the new findings are consistent with the team’s prior research on which brain centers contribute most to schizophrenia in adults suggests there may be a way to prevent it, he said. “In schizophrenia, by the time of diagnosis, a lot of damage has already occurred in the brain, and it can be very difficult to change the course of the disease.”

“What we saw is that, early on, functional interactions among brain regions within the same brain systems are abnormal,” he added. “The abnormalities do not start when you are in your 20s; they are evident even when you are 7 or 8.”

Our discoveries underscore the importance of approaching people with psychosis with compassion.

The researchers plan to use existing treatments, such as transcranial magnetic stimulation or focused ultrasound, targeted at these brain centers in young people at risk of psychosis, such as those with 22q11.2 deletion syndrome or with two parents who have schizophrenia, to see if they prevent or delay the onset of the condition or lessen symptoms once they appear. 

The results also suggest that using functional MRI to monitor brain activity at the key centers could help scientists investigate how existing antipsychotic medications are working. 

Although it’s still puzzling why someone becomes untethered from reality — given how risky it seems for one’s well-being — the “how” is now understandable, Supekar said. “From a mechanistic point of view, it makes sense,” he said.

“Our discoveries underscore the importance of approaching people with psychosis with compassion,” Menon said, adding that his team hopes their work not only advances scientific understanding but also inspires a cultural shift toward empathy and support for those experiencing psychosis. 

“I recently had the privilege of engaging with individuals from our department’s early psychosis treatment group,” he said. “Their message was a clear and powerful: ‘We share more similarities than differences. Like anyone, we experience our own highs and lows.’ Their words were a heartfelt appeal for greater empathy and understanding toward those living with this condition. It was a call to view psychosis through a lens of empathy and solidarity.”

Researchers contributed to the study from UCLA, Clinica Alemana Universidad del Desarrollo, Pontificia Universidad Católica de Chile, the University of Oxford and UC Davis.

The study was funded by the Stanford Maternal and Child Health Research Institute’s Uytengsu-Hamilton 22q11 Neuropsychiatry Research Program, FONDEYCT (the National Fund for Scientific and Technological Development of the government of Chile), ANID-Chile (the Chilean National Agency for Research and Development) and the U.S. National Institutes of Health (grants AG072114, MH121069, MH085953 and MH101779).

About Stanford Medicine

Stanford Medicine is an integrated academic health system comprising the Stanford School of Medicine and adult and pediatric health care delivery systems. Together, they harness the full potential of biomedicine through collaborative research, education and clinical care for patients. For more information, please visit med.stanford.edu .

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Moscow’s new suburban rail lines dramatically improve commutes!

The first two of the five lines of the MCD were launched November 21: the MCD1 line goes to Odintsovo (west) and Lobnya (north), while MCD2 to Podolsk (south) and Nakhabino (west). These towns are some 20-30 km from Moscow, and are inhabited by hundreds of thousands of people traveling to Moscow on business each day.

Vladimir Putin at the opening of the MCD.

Both lines cross over with central Moscow stations, as well as those on the outskirts. You can change over to the Moscow Central Circle or the metro (there is a total of 27 stations where you can make the switch). These changes allow for getting to and from the city center much faster and more conveniently than commuter trains.

Why does Moscow need the MCD?

The MCD will add to the function of the commuter trains, easing part of the daily burden experienced by those lines, as well as reduce car traffic in and out of town. The MCD runs every 5-10 minutes with only a four-hour break at night. The reason the new trains are referred to as Diameters is that they travel not just to Moscow’s main intercity stations, but also from one city on the outskirts of Moscow all the way to another one situated at the other end. 

The service runs similar to an ordinary Metro, from 5:30 a.m. to 1 a.m. Directions are available in English, so you won’t get lost.

One important change you must remember is the Aeroexpress trains to Sheremetyevo airport now run differently than before: now one ride from the Belorussky railway to Sheremetyevo will take 50 minutes - instead of the usual 35. Also, on the way from the airport, Belorussky railway will no longer be the final stop - Odintsovo will. But there will also be a few new stations that cross over with the regular metro - in the Moscow City district, on Begovaya, Fili and Kuntsevskaya. All of these interlink with the regular metro.

New internet-capable trains

The MCD trains - in accordance with the Russian custom with giving names of birds to every new train route - will carry the name Ivolga (Russian word for “Eurasian golden oriole”). The carriages are outfitted with bike racks, luggage and stroller compartments, power sockets and USB chargers with every seat, as well as free Wi-Fi - as is the case with all of Moscow’s urban transport. 

39 trains are currently operational.

Free transfers!

Prior to the MCD appearing, passengers wanting to switch between city and suburban trains had to buy separate tickets. The MCD greatly simplifies the process: now you simply need to use your Troika card at the MCD ticket stalls. There are two tariffs - city (38 rubles, or $0.6) and regional (45 rubles, $0.8; meaning cities just on the outskirts of Moscow - not the entire region). If you need to go further afield into the Moscow region, beyond the MCD, then your Troika card will be charged an additional $0.35 (23 rubles) to transfer to the ‘elektrichka’ (electrical commuter trains). 

Switching between MCD and metro trains is free for a 90-minute window, irrespective of the tariff. However, if you don’t check out of the station at the exit stalls, your Troika card will be blocked, and require around $2.30 (150 rubles) to unlock. 

You can also pay via Apple Pay, Google Pay, Mastercard PayPass and Visa PayPass. 

Will there be other ‘diameters’?

Several more MCD are slated to launch before 2025 - the MCD3 (from Ramenskoye to Zelenograd), MCD4 (Zheleznodorozhny to Aprelevka) and MCD5 (from Domodedovo to Pushkino). Right now, the passenger load of the first two Diameters is estimated at 900,000 per day. All five Diameters will serve more than 330 million Moscow passengers every year. 

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While Saturday felt wintry to some in the Northeast, a quick-moving storm system that will drop out of the Great Lakes has a chance of producing showers and thunderstorms on Sunday.

The FOX Forecast Center expects some thunderstorms to develop during the late morning and afternoon as they work from northwest to southeast across the region.

The Storm Prediction Center has highlighted communities from the eastern Ohio Valley through western Massachusetts for being at risk of strong to severe storms.

Communities such as State College and Scranton in Pennsylvania are included in NOAA’s Level 3 out of 5 severe storm threat zone.

Damaging winds are expected to be the primary hazard, but a brief tornado and hail are also possible in the strongest of the storms.

Approximately 34 million are under the ‘possible’ threat zone on Sunday, with 2 million in the ‘very likely’ region.

Fortunately for the I-95 corridor, the threat of damaging thunderstorms is expected to greatly decrease after sunset, as storms run out of energy.

Cities such as Philadelphia, New York and Boston sit on the edge or just outside the ‘possible’ threat zone for severe weather.

Depending on where the initial round of showers and thunderstorms develop, additional precipitation could fall over the eastern Ohio River Valley.

Although significant flash flooding is not expected, dozens of water gauges along the Ohio River and its tributaries are at flood stage and any additional rainfall will aggravate water levels.

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